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In Guillain-Barré Syndrome (GBS), it is important to note that the condition often follows a recent infection, particularly viral or bacterial, which triggers an autoimmune response. This response can lead to the demyelination of peripheral nerves and result in the characteristic weakness and sensory loss associated with GBS.

In particular, certain viral infections, such as those caused by the cytomegalovirus, Epstein-Barr virus, or Zika virus, as well as bacterial infections like Campylobacter jejuni, have been prominently associated with the onset of GBS. This infection history is crucial in understanding the patient’s condition, as it illustrates how GBS can follow a pathophysiological sequence that begins with an infectious agent.

While vaccinations can sometimes be implicated in GBS cases, the incidence in relation to recent infections is more significantly documented in the medical literature. Moreover, not all vaccines are associated with an increased risk, and the overall risk remains exceedingly rare in relation to the number of vaccinations administered. Therefore, Recent infection is a more consistent and commonly noted event in the history of patients with Guillain-Barré Syndrome.